Great News on SARS-CoV-2 - T-Cell "Memory" and 100 Monkey Spread Via The Collective Unconscious

stock here, this re-blogged from Majia's blog, a great anti-nuclear educator and author of several
books.    LOL a Liberal who can still tolerate me.

I sent in my hypothesis about the human collective unconcious being able to program a species wide production of T-cells, at about 10% population exposure.

Majia did some digging and came up with this very interesting scenario. 

At first I was concerned it shut down my pet hypothesizes.    But I realized....we won't have species wide T-cell memory, but if enough people in the 10% have T Cell memory, then like the 100 Monkey Thing, that knowledge can spread to the whole species.

OP
https://www.nature.com/articles/s41586-020-2550-z

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Recently the news on COVID-19 has been mostly bad as the evidence has grown for serious and lasting bodily damage among many recovered patients.  Additionally, the news on antibodies has been disappointing, raising the risk of never-ending re-infections, which will be extremely difficult to prevent if the virus spreads effectively in aerosols.

But the good news is GOOD! Memory T cells to our rescue!!!!!!!!!!!!!

It seems we have some hidden immunity beyond antibodies.

It turns out that our T cells can remember and react to coronavirus attacks, including from the viruses that cause SARS and COVID-19, as this new study published at Nature reports in its abstract:

Le Bert, N., Tan, A.T., Kunasegaran, K. et al. SARS-CoV-2-specific T cell immunity in cases of COVID-19 and SARS, and uninfected controls. Nature (2020). https://doi.org/10.1038/s41586-020-2550-z. https://www.nature.com/articles/s41586-020-2550-z

Memory T cells induced by previous pathogens can shape the susceptibility to, and clinical severity of, subsequent infections1. Little is known about the presence of pre-existing memory T cells in humans with the potential to recognize SARS-CoV-2.

Here, we first studied T cell responses to structural (nucleocapsid protein, NP) and non-structural (NSP-7 and NSP13 of ORF1) regions of SARS-CoV-2 in COVID-19 convalescents (n=36). In all of them we demonstrated the presence of CD4 and CD8 T cells recognizing multiple regions of the NP protein.

We then showed that SARS-recovered patients (n=23) still possess long-lasting memory T cells reactive to SARS-NP 17 years after the 2003 outbreak, which displayed robust cross-reactivity to SARS-CoV-2 NP.

Surprisingly, we also frequently detected SARS-CoV-2 specific T cells in individuals with no history of SARS, COVID-19 or contact with SARS/COVID-19 patients (n=37). SARS-CoV-2 T cells in uninfected donors exhibited a different pattern of immunodominance, frequently targeting the ORF-1-coded proteins NSP7 and 13 as well as the NP structural protein. Epitope characterization of NSP7-specific T cells showed recognition of protein fragments with low homology to “common cold” human coronaviruses but conserved amongst animal betacoranaviruses.

Thus, infection with betacoronaviruses induces multispecific and long-lasting T cell immunity to the structural protein NP. Understanding how pre-existing NP- and ORF-1-specific T cells present in the general population impact susceptibility and pathogenesis of SARS-CoV-2 infection is of paramount importance for the management of the current COVID-19 pandemic.

This is really good news.
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